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Chapter 15 ANALGESICA AND ANESTHESIA - page 27 / 52

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ANESTHETIC AGENTS

CARDIOVASCULAR TOXICITY

Do not always follow CNS involvement

Develop later than those from cerebral toxicity ← induced by higher blood levels of drug

Characterized first by stimulation and then depression

Hypertension & tachycardia → hypotension & cardiac arrhythmias

Impaired U-P perfusion & fetal distress

Turning the woman onto either side to avoid aortocaval compression

Crystalloid solution : infused rapidly, IV ephedrine

Emergency c/sec : maternal vital signs have not been restored within 5 min of cardiac arrest

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