and mashed potatoes. You can figure out portion sizes of various combinations of foods containing coconut and coconut oil equivalent to at least 35 grams of fat from coconut oil.
If you are using any type of hydrogenated veg- etable oil or any oil with transfat, do not use any more and get rid of it! Extra virgin olive oil, but- ter and other natural, non-hydrogenated oils are okay to use along with the coconut oil. It is possible to use coconut oil in place of all other oils, how- ever, since it contains no omega-3 fatty acids, it is very important to eat salmon twice a week or get enough omega-3 fatty acid from other rich sources such as fish oil capsules, flax meal, flax oil (not for cooking) or walnuts.
It is inconceivable that a potential dietary pre- vention and cure for Alzheimer’s disease, and other neurodegenerative diseases, has been out there for so many years, and yet has gone unnoticed. It is very likely that these diseases are becoming more prevalent due our current diet. The American diet has changed drastically from what it was before the 1950’s, when our parents and grandparents used lard and coconut oil to cook. Cardiovascular dis-
ease was rare at the beginning of the 20th century, and has skyrocketed, along with other devastating diseases, such as Alzheimer’s, diabetes type II, obe- sity, since mass produced hydrogenated vegetable oils containing trans fats were introduced into our diets and replaced these other natural fats. Sadly, the incidences of cardiovascular and other serious diseases are becoming more and more common among people in other areas of the world who have changed over from their indigenous foods to the “western” diet.
I plan to tell everyone I can and get this infor- mation to persons in positions to investigate this with the hope that Dr. Veech and other MCT oil and ketone body researchers get the funding they need. Feel free to make copies and pass this write- up on.
If you have a loved one or a patient with Alzheimer’s or one of these other degenerative neurologic diseases, consider trying coconut oil. Dr. Veech suggests that, if possible, a videotape of the person before starting and at various points af- ter starting the coconut oil would be very useful to document change. He suggests including segments
of the persons face, speech and gait (walking). He also advises to have ketone bodies measured. What have you got to lose?
Dr. Mary Newport 10030 Orchard Way Spring hill, FL 34608 Home: (352) 666-1025 Cell: (352) 428-0251 Preemiedoctor@aol.com
Coconut oil and MCT oil websites: www.coconutoilresearch.com www.nutiva.com • www.amazon.com www.tropicaltraditions.com www.oilsbynature.com www.cheapvitamins.com Palm kernel oil website: www.oilsbynature.com Coconut oil and coconut milk are also avail- able at most health food stores and many grocery stores.
“Ketone bodies, potential therapeutic uses,” RL Veech, B Chance, Y Kashiwaya, HA Lardy, GC Cahill, Jr., IUBMB Life, 2001, Vol. 51 No.4, 241-247
“Ketoacids? Good Medicine?” George F. Cahill, Jr., Richard L. Veech, Transactions of the American Clinical and Climatological Association, Vol. 114, 2003.
“The therapaeutic implications of ketone bodies: the effects of ketone bodies in pathological conditions: ketosis, ketogenic diet, redox states, insulin resistance, and mitochondrial metabolism,” Richard L. Veech, Prostaglandins, Leukotrienes and Essential Fatty Acids, 70 (2004) 309-319.
“Diminished glucose transport and phosphorylation in Alzheimer’s Disease determined by dynamic FDG-PET,” M Piert, et.al., The Journal of Nuclear Medicine, Vol.37 No.2, February 1996, 201-208.
“Glucose metabolism in early onset versus late onset Alzheimer’s Disease: an SPM analysis of 120 patients,” EJ Kim, et. al., Brain, 2005, Vol. 128, 1790-1801.
“Cerebral glucose metabolism in Parkinson’s disease with and without dementia,” RF Peppard, et.al., Archives of Neurology, Vol. 49 No.12, December 1992.
“Cortical and subcortical glucose consumption measured by PET in patients with Huntington’s disease,” Brain, October 1990, Vol 113, part 5, 1405-23.
“Reduced glucose metabolism in the frontal cortex and basal ganglia of multiple sclerosis patients with fatigue: a 18F-fluorodeoxyglucose positron emission tomography study,” U Roelcke, et. al., Neurology, 1997, Vol. 48, Issue 6, 1566-1571.
“ALS-linked Cu/Zn-SOD mutation impairs cerebral synaptic glucose and glutamate transport and exacerbates ischemic brain injury,” Z Guo, et. al., Journal of Cerebral Blood Flow Metabolism, March 2000, Vol. 20 No. 3, 463-8.
“Combinations of medium chain triglycerides and therapeutic agents for the treatment and prevention of Alzheimer’s disease and other diseases resulting from reduced neuronal metabolism,” United States Patent 20080009467, Inventor Samuel T. Henderson, Accera, Inc., Broomfield, Colorado (Ketasyn).
Nutrient analysis of coconut oil (vegetable), NDB No: 04047 – www.nal.usda.gov/fnic/foodcomp .
“Lipids in (human) milk and the first steps in their digestion,” M Hamosh, et. al., Pediatrics, 1985, Vol. 75, 146-150.
“Nutritional factors and serum lipid levels,” EH Ahrens, American Journal of Medicine, 1957, vol. 23, 928 (used hydrogenated coconut oil).
“Trans fatty acids and coronary artery disease,” NEJM, 1999, Vol. 340, 1994-1998.
“Effect of mixed fat formula feeding on serum cholesterol level in man,” SA Hashim, American Journal of Clinical Nutrition, 1959, Vol. 7, 30-34.
“Modified-fat dietary management of the young male with coronary disease: a five-year report,” JL Bierenbaum, JAMA, 1967, Vol. 202, 1119-1123.
“Cholesterol, coconuts and diet in Polynesian atolls-a natural experiment; the Pukapuka and Toklau island studies,” IA Prior, American Journal of Clinical Nutrition, 1981, Vol. 34, 1552-1561.
“Changes in cerebral blood flow and carbohydrate metabolism during acute hyperketonemia,” S.G. Hasselbalch, et.al, Am J Physiol, 1996, Vol. 270, E746-51.
“Effect of hyperketonemia and hyperlacticacidemia on symptoms, cognitive dysfunction, and counterregulatory hormone responses during hypogly- cemia in normal humans,” T. Veneman, et. al., Diabetes 43:1311-7 (1994).
“D-b-Hydroxybutyrate protects neurons in models of Alzheimer’s and Parkinson’s disease,” Y Kashiwaya, et. al. including RL Veech, PNAS, May 9, 2000, Vol. 97 No. 10, 5440-5444.
“High carbohydrate diets and Alzheimer’s disease,” Samuel T. Henderson, Medical Hypotheses, 2004, Vol 62, 689-700 (Another article of interest).
“Effects of b-Hydroxybutyrate on cognition in memory-impaired adults,” MA Reger, ST Henderson, et. al., Neurobiology of Aging, 2004, Vol. 25, 311-314.
“Breastfeeding, infant formula supplementation, and Autistic Disorder: the results of a parent survey,” ST Schultz, et. al., International Breastfeeding Journal, 2006, Vol. 1 No. 16.
Other Important Resources “Ketones: Metabolism’s Ugly Duckling,” TB VanItallie, TH Nufert, Nutrition Reviews, Vol 61, No 10, 327-341. “Fuel Metabolism in Starvation,” GF Cahill, Jr., Annual Reviews in Nutrition, 2006, 26:1-22. “Ketone Bodies as a Therapeutic for Alzheimer’s Disease,” ST Henderson, Journal of the American Society for Experimental NeuroTherapeutics,
Vol 5, 470-480, July 2008.